Thoracic and abdominal injury outcomes are correlated to chestband-derived uniaxial metrics in post-mortem human specimen (PMHS) experiments. Yet, it is desirable to establish a relationship between localized deformation behaviors and tissue response throughout the impact event. Using 2D chestband contours from PMHS experiments, visceral strain and strain energy density responses were examined using a planar viscoelastic finite element model developed from NLM Visible Human Project images at the T11 level. Mesh geometry consisted of vertebra, chest wall, liver, spleen, omentum/hollow viscera, and external tissue. Material properties were obtained from literature. Validation compared model to published loading responses. The model was exercised by applying to the model periphery 21 subject-specific PMHS chestband deformation patterns; material responses in the spleen were determined. Chestband contour data represented four impact boundary conditions: (a) lateral impact with close-proximity torso airbag (n=4), (b) stationary close-proximity torso airbag loading (n=3), (c) flat rigid lateral impact (n=7), and (d) anterolateral oblique rigid impact (n=4). Mean peak localized spleen strain responses for boundary conditions (a)-(d) were: (a) 2.26, (b) 0.68, (c) 1.24, and (d) 0.83. Mean peak strain energy density responses were: (a) 38.2, (b) 6.0 (c) 17.4, and (d) 5.6 kJ/m3. ANOVA determined these results to be significant (p < 0.002). Using matched-pair experiment injuries, 50% risk of visceral trauma corresponded to localized strain and strain energy density of 1.55 and 33.0 kJ/m3 (p < 0.1). Although strains were large, model response demonstrated sensitivity to impact boundary conditions and observed PMHS trauma. This model formulation is useful for examining injury risk from torso deformations measured experimentally using the chestband device.